Condition Guide Updated May 2026 9 min read

Red Light Therapy for Gluteal Tendinopathy: Reducing Lateral Hip Pain

Gluteal tendinopathy — formerly mislabelled as "trochanteric bursitis" — is the most common cause of lateral hip pain in adults over 40. It's a tendon problem, not a bursa problem, and tendinopathy responds well to photobiomodulation.

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If you have a sharp or aching pain on the outside of your hip that worsens when you lie on it, climb stairs, or stand from a low chair, you're probably dealing with gluteal tendinopathy. It affects up to 23% of women and 8% of men aged 50 and over, and is the leading non-surgical cause of lateral hip disability in this group.

For decades it was misdiagnosed as "trochanteric bursitis" — an inflamed bursa over the bony point of the hip. Modern imaging has shown that the bursa is rarely the primary problem. The actual pathology is degeneration and inflammation of the gluteus medius and gluteus minimus tendons where they attach to the greater trochanter. That distinction matters because it changes the treatment: tendinopathy responds to load management and tissue support, not to anti-inflammatory injections aimed at the bursa.

What Is Gluteal Tendinopathy?

The gluteus medius and gluteus minimus are two of the deeper hip stabilising muscles. Both attach to the greater trochanter — the bony prominence on the outside of the hip. These tendons take huge cumulative load: every step you take, every time you stand on one leg, the gluteal tendons fire to keep the pelvis level.

Tendinopathy is what happens when the tendon's normal repair processes fall behind the load being placed on it. The tendon develops microdamage, the collagen architecture gets disorganised, neurovascular ingrowth occurs, and pain becomes chronic. The condition is most common in middle-aged and older women — partly due to hormonal changes affecting tendon collagen, partly due to hip biomechanics that load the lateral tendons more in women than men.

The classic presentation: pain on the outside of the hip, worse when you lie on the affected side at night, worse with stairs and prolonged standing, often radiating into the lateral thigh. There's almost always tenderness directly over the greater trochanter when pressed.

Why It's Often Misdiagnosed as Bursitis

For decades, lateral hip pain was treated as "trochanteric bursitis" — an inflammation of the bursa sitting over the greater trochanter. Patients were given corticosteroid injections aimed at the bursa, with mixed results. Modern ultrasound and MRI imaging has shown that in the vast majority of cases, the bursa is normal or only mildly involved. The actual problem is in the tendons.

This is why corticosteroid injection results are inconsistent — they're sometimes hitting the wrong tissue. It's also why the condition is now formally called "greater trochanteric pain syndrome" (GTPS) or "gluteal tendinopathy" in physiotherapy and sports medicine literature, not bursitis.

For photobiomodulation, the distinction matters because tendinopathy has a much better evidence base for LLLT than bursitis does. The mechanism — reducing inflammatory signalling, supporting collagen organisation, addressing the cellular environment around chronic tendon damage — maps directly onto what tendinopathy actually is.

What the Research Shows

The strongest LLLT evidence in tendinopathy comes from Achilles, patellar, rotator cuff, and elbow studies — the gluteal tendons specifically have less direct evidence, but the mechanism is the same. A 2024 meta-analysis of tendon outcomes found that LLLT reduces pain intensity and may improve function in tendon conditions when applied at clinically meaningful doses.

For lateral hip pain specifically, current best practice combines load management (avoiding sleeping on the affected side, modifying stair use, single-leg stance work) with progressive isometric and isotonic strengthening of the hip abductors. Photobiomodulation is increasingly used as an adjunct to support tendon recovery between exercise sessions, particularly during the first 8–12 weeks when symptoms are most active.

A 2021 review of LLLT in soft-tissue rehabilitation concluded that LLLT is a beneficial adjunct to rehabilitation exercise — the same pattern that holds for knee OA holds for tendinopathy.

How to Use the MOVE+ for Gluteal Pain

Application is straightforward — the gluteal tendons are surface-accessible at the greater trochanter, and 808 nm penetrates easily to the tendon depth.

Suggested protocol:

  • Placement: wrap directly over the greater trochanter (the bony point on the outside of the hip — you'll feel it easily) and the area immediately above and behind it.
  • Wavelength: 660 nm + 808 nm (MOVE+ standard).
  • Session duration: 10–15 minutes per session.
  • Frequency: 5 sessions per week for the first 8 weeks, then taper to 3 sessions per week.
  • Combine with: isometric hip abductor work (side-lying clamshells, side-lying leg lifts at low load), single-leg balance work, and load management (avoid sleeping on the affected side; use a pillow between knees if you do).
  • Avoid: aggressive stretching of the iliotibial band — for gluteal tendinopathy, ITB stretching often makes the condition worse.

When to Combine With PT vs. When to Consider Injection

Most cases of gluteal tendinopathy improve with 8–12 weeks of structured exercise rehabilitation plus daily photobiomodulation. If symptoms are severe at presentation or aren't improving after 12 weeks of disciplined conservative care, options include image-guided high-volume injection (saline + small steroid dose), platelet-rich plasma (PRP), or — in refractory cases — surgical tendon repair.

Photobiomodulation pairs particularly well with PT because it reduces the post-exercise inflammatory load that often causes tendinopathy patients to flare up after rehab sessions. Many physiotherapists now incorporate LLLT into clinic visits for exactly this reason.

Safety and Contraindications

  • Do not apply directly over recent injection sites for at least 48 hours.
  • Avoid use over active infection or open skin lesions.
  • Pregnancy: see our pregnancy guide.
  • Photosensitising medications (tetracyclines, isotretinoin, some NSAIDs at high doses): consult your prescriber.
  • Hip implants: photobiomodulation is non-thermal and does not interact with metal hardware, but always confirm with your orthopaedic surgeon.

Frequently Asked Questions

No — and this is one of the most consistent misdiagnoses in lateral hip pain. The historical label "trochanteric bursitis" implied an inflamed bursa, but modern imaging studies show the bursa is rarely the primary issue. The actual pathology is tendinopathy of the gluteus medius and minimus tendons. The condition is now formally called gluteal tendinopathy or greater trochanteric pain syndrome (GTPS) in current sports medicine and physiotherapy literature.
Most cases improve substantially within 8–12 weeks of disciplined load management plus progressive strengthening, with daily photobiomodulation as an adjunct. Recovery is rarely linear — symptoms often improve in waves. Persistent severe symptoms beyond 12 weeks of consistent conservative care may warrant imaging and consideration of injection or PRP.
Side-lying compresses the gluteal tendons against the greater trochanter and the underside of the iliotibial band. The mechanical compression irritates the already-sensitive tendon. Use a pillow between your knees and try to sleep on the unaffected side; some users find a small folded towel under the affected hip when supine helps reduce the angle of stretch.
Generally no — for gluteal tendinopathy, aggressive ITB stretching typically makes the condition worse by increasing compressive load on the tendons. Focus instead on isometric strengthening of the hip abductors, gentle hip external rotator work, and posterior chain mobility. If you have access to a physiotherapist, get specific guidance on which mobility work helps and which makes things worse.
Yes — once the immediate post-injection period has passed (typically 48 hours), photobiomodulation can be added safely. In fact, LLLT may help support tendon health while the corticosteroid effect wears off. Some clinicians are increasingly cautious about repeated steroid injections into tendinopathic tissue because of potential effects on tendon collagen; LLLT can be a useful complement that doesn't carry that concern.
Yes — it's significantly more common in women (roughly 23% prevalence in women over 50 vs 8% in men), but men aren't immune. Male presentation tends to be more associated with running and athletic loading patterns; female presentation tends to be more associated with hip biomechanics, hormonal effects on tendon collagen, and post-menopausal changes. The treatment principles are the same in either case.

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About the Author

CB
Chris Bohler Chief Technology Officer, Kineon

Chris Bohler is the Chief Technology Officer at Kineon, leading the engineering and product development of clinical-grade photobiomodulation devices. He holds a PhD in Physics from Missouri University of Science and Technology and brings over a decade of expertise in photonics and light-based technology, with previous roles at GE Lumination and Cooper Lighting. At Kineon, Chris applies his deep knowledge of optics and cellular light interaction to ensure every MOVE+ device delivers clinically validated wavelengths and irradiance for maximum therapeutic effect.

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Key Referenced Researchers

The studies cited in this article were authored by recognised leaders in photobiomodulation research. Below is a brief overview of the principal investigators whose work forms the evidence base for this guide.

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Pinar Avci, MD Researcher · Wellman Center for Photomedicine, Harvard Medical School / Massachusetts General Hospital

Dr. Avci conducted her photobiomodulation research at the Wellman Center for Photomedicine at Harvard Medical School alongside Dr. Michael Hamblin. Her work covers the mechanisms and applications of light therapy across skin, musculoskeletal, and joint conditions, contributing to the preclinical and clinical evidence base for PBM in pain and inflammatory disorders.

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Ernesto Cesar Pinto Leal-Junior, PhD Full Professor · Nove de Julho University, São Paulo · Associate Professor, University of Bergen

Dr. Leal-Junior has authored over 140 peer-reviewed publications in photobiomodulation — more randomised controlled trials than any other researcher in the field. His research spans sports performance, muscular fatigue, tendinopathy, and post-exercise recovery. Supported by USD 3M+ in grants, he leads the Laboratory of Phototherapy and Innovative Technologies in Health (LaPIT) in Brazil.

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Michael R. Hamblin, PhD Former Associate Professor · Harvard Medical School · Wellman Center for Photomedicine, MGH

Dr. Hamblin is one of the world's foremost authorities on photobiomodulation, with over 720 peer-reviewed publications, an h-index of 143, and more than 80,000 citations. As Principal Investigator at the Wellman Center for Photomedicine, Massachusetts General Hospital, his research established the foundational cellular mechanisms by which red and near-infrared light modulates inflammation, accelerates tissue repair, and supports neural recovery.

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